Pressure Overload–Mediated Sustained PKR2 (Prokineticin-2 Receptor) Signaling in Cardiomyocytes Contributes to Cardiac Hypertrophy and Endotheliopathies
Ferda Demir(Inserm), Canan G. Nebigil(Inserm), Hitoshi Kurose(Tokushima University), Anaïs Audebrand(Inserm), Marja Steenman(Inserm), Kyoji Urayama(Kyushu University), Ayça Toprak Semiz(Inserm)
Cited by 6
Related Papers
LTRPC2 Ca2+-Permeable Channel Activated by Changes in Redox Status Confers Susceptibility to Cell Death
|Molecular Cell|2002|830
Specific uncoupling by islet-activating protein, pertussis toxin, of negative signal transduction via alpha-adrenergic, cholinergic, and opiate receptors in neuroblastoma x glioma hybrid cells.
|Journal of Biological Chemistry|1983|497
TRPC3 and TRPC6 are essential for angiotensin II‐induced cardiac hypertrophy
|The EMBO Journal|2006|417
Selective and direct inhibition of TRPC3 channels underlies biological activities of a pyrazole compound
|Proceedings of the National Academy of Sciences|2009|387