LTRPC2 Ca2+-Permeable Channel Activated by Changes in Redox Status Confers Susceptibility to Cell Death
Yuji Hara(University of Shizuoka), Yasuo Mori(Kyushu University), Masakazu Ishii(Okazaki Institute for Integrative Bioscience), Yasunobu Okada(Aichi Medical University), Keiji Imoto(National Institutes of Natural Sciences), Shunichi Shimizu(SHOWA Medical University), Motohiro Nishida(Kyushu University), Minoru Wakamori(Okazaki Institute for Integrative Bioscience), Hisanobu Yamada(Okazaki Institute for Integrative Bioscience), Emi Maeno(The Graduate University for Advanced Studies, SOKENDAI), Takashi Yoshida(Hirosaki University), Emiko Mori(Okazaki Institute for Integrative Bioscience), Hitoshi Kurose(Tokushima University), Nobuyoshi Shimizu(Chiba-Nishi General Hospital), Jun Kudoh(Keio University)
Cited by 830
Related Papers
Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase
|Nature Genetics|2000|2k
Primary structure and functional expression from complementary DNA of a brain calcium channel
|Nature|1991|869
A Unified Nomenclature for the Superfamily of TRP Cation Channels
|Molecular Cell|2002|679