Patterns of transcription factor programs and immune pathway activation define four major subtypes of SCLC with distinct therapeutic vulnerabilities
Carl M. Gay(The University of Texas MD Anderson Cancer Center), C. Allison Stewart(The University of Texas MD Anderson Cancer Center), Elizabeth M. Park(The University of Texas MD Anderson Cancer Center), Lixia Diao(The University of Texas MD Anderson Cancer Center), Sarah M. Groves(Vanderbilt University Medical Center), Simon Heeke(The University of Texas MD Anderson Cancer Center), Barzin Y. Nabet, Junya Fujimoto(The University of Texas MD Anderson Cancer Center), Luisa M. Solis(The University of Texas MD Anderson Cancer Center), Wei Lü(The University of Texas MD Anderson Cancer Center), Yuanxin Xi(The University of Texas MD Anderson Cancer Center), Robert J. Cardnell(The University of Texas MD Anderson Cancer Center), Qi Wang(The University of Texas MD Anderson Cancer Center), Giulia Fabbri(AstraZeneca (United States)), Kasey R. Cargill(The University of Texas MD Anderson Cancer Center), Natalie I. Vokes(The University of Texas MD Anderson Cancer Center), Kavya Ramkumar(The University of Texas MD Anderson Cancer Center), Bingnan Zhang(The University of Texas MD Anderson Cancer Center), Carminia Maria Della Corte(University of Campania "Luigi Vanvitelli"), Paul Robson(Jackson Laboratory), Stephen G. Swisher(The University of Texas MD Anderson Cancer Center), Jack A. Roth(The University of Texas MD Anderson Cancer Center), Bonnie S. Glisson(The University of Texas MD Anderson Cancer Center), David S. Shames, Ignacio I. Wistuba(The University of Texas MD Anderson Cancer Center), Jing Wang(The University of Texas MD Anderson Cancer Center), Vito Quaranta(Vanderbilt University Medical Center), John D. Minna(The University of Texas Southwestern Medical Center), John V. Heymach(The University of Texas MD Anderson Cancer Center), Lauren A. Byers(The University of Texas MD Anderson Cancer Center)
Cited by 919Open Access
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