Zmat3 Is a Key Splicing Regulator in the p53 Tumor Suppression Program
Kathryn Bieging-Rolett(Stanford University), Laura D. Attardi(Stanford University)
Cited by 119
Related Papers
Global genomic profiling reveals an extensive p53-regulated autophagy program contributing to key p53 responses
|Genes & Development|2013|446
<i>Neat1</i> is a p53-inducible lincRNA essential for transformation suppression
|Genes & Development|2017|204
Pathways connecting telomeres and p53 in senescence, apoptosis, and cancer
|Biochemical and Biophysical Research Communications|2005|191
Multifaceted role for p53 in pancreatic cancer suppression
|Proceedings of the National Academy of Sciences|2023|43