<i>Neat1</i> is a p53-inducible lincRNA essential for transformation suppression
Stephano S. Mello(University of Rochester Medical Center), Laura D. Attardi(Stanford Medicine)
Cited by 204
Related Papers
Global genomic profiling reveals an extensive p53-regulated autophagy program contributing to key p53 responses
|Genes & Development|2013|446
Zmat3 Is a Key Splicing Regulator in the p53 Tumor Suppression Program
|Molecular Cell|2020|119
Multifaceted role for p53 in pancreatic cancer suppression
|Proceedings of the National Academy of Sciences|2023|43
Puma- and Caspase9-mediated apoptosis is dispensable for p53-driven neural crest-based developmental defects
|Cell Death and Differentiation|2021|9