Behavioral and neural network abnormalities in human APP transgenic mice resemble those of App knock-in mice and are modulated by familial Alzheimer’s disease mutations but not by inhibition of BACE1
Erik C. B. Johnson(Emory University), Lennart Mucke(Gladstone Institutes), Pascal E. Sanchez(Denali Therapeutics (United States)), Biljana Djukic(Gladstone Institutes), Jeanne T. Paz(Gladstone Institutes), Jorge J. Palop(Gladstone Institutes), Weiping Zhang(University of Illinois Urbana-Champaign), Gui-Qiu Yu(Gladstone Institutes), Michael Gill(Gladstone Institutes), Melanie Das(Cedars-Sinai Medical Center), Kaitlyn Ho(Gladstone Institutes), Xinxing Yu(Gladstone Institutes)
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