Restoration of histone acetylation ameliorates disease and metabolic abnormalities in a FUS mouse model

Elisabeth Rossaert(Allen Institute for Brain Science), Ludo Van Den Bosch(VIB-KU Leuven Center for Brain & Disease Research)

Acta Neuropathologica Communications

July 5, 2019

10.1186/s40478-019-0750-2

Cited by 89

Related Papers

HDAC6 inhibition reverses axonal transport defects in motor neurons derived from FUS-ALS patients

|Nature Communications|2017|377

Disrupted function and axonal distribution of mutant tyrosyl-tRNA synthetase in dominant intermediate Charcot-Marie-Tooth neuropathy

|Nature Genetics|2006|376

HDAC6 is a therapeutic target in mutant GARS-induced Charcot-Marie-Tooth disease

|Brain|2017|120

<i>C9orf72</i> -derived arginine-containing dipeptide repeats associate with axonal transport machinery and impede microtubule-based motility

|Science Advances|2021|90

CRISPR/Cas9 screen in human iPSC‐derived cortical neurons identifies NEK6 as a novel disease modifier of <i>C9orf72</i> poly(PR) toxicity

|Alzheimer s & Dementia|2022|26