Immune evasion mediated by PD-L1 on glioblastoma-derived extracellular vesicles
Franz Ricklefs(Brigham and Women's Hospital), Quazim A. Alayo(Brigham and Women's Hospital), Harald Krenzlin(Brigham and Women's Hospital), Ahmad Bakur Mahmoud(Brigham and Women's Hospital), Maria C. Speranza(Brigham and Women's Hospital), Hiroshi Nakashima(Brigham and Women's Hospital), Josie Hayes(University of California, Berkeley), Kyungheon Lee(Massachusetts General Hospital), Leonora Balaj(Massachusetts General Hospital), Carmela Passaro(Brigham and Women's Hospital), Arun K. Rooj(Brigham and Women's Hospital), Susanne Krasemann(Universität Hamburg), Bob S. Carter(Massachusetts General Hospital), Clark C. Chen(University of California San Diego), Tyler Steed(University of California San Diego), Jeffrey M. Treiber(University of California San Diego), Scott J. Rodig(Brigham and Women's Hospital), Katherine S. Yang(Brigham and Women's Hospital), Ichiro Nakano(University of Alabama at Birmingham), Hakho Lee(Massachusetts General Hospital), Ralph Weissleder(Massachusetts General Hospital), Xandra O. Breakefield(Massachusetts General Hospital), Jakub Godlewski(Brigham and Women's Hospital), Manfred Westphal(Universität Hamburg), Katrin Lamszus(Universität Hamburg), Gordon J. Freeman(Harvard University), Agnieszka Bronisz(Brigham and Women's Hospital), Sean Lawler(Brigham and Women's Hospital), E. Antonio Chiocca(Brigham and Women's Hospital)
Cited by 608Open Access
Abstract
. These results suggest that PD-L1 on EVs may be another mechanism for glioblastoma to suppress antitumor immunity and support the potential of EVs as biomarkers in tumor patients.
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