Cross-disorder risk gene CACNA1C differentially modulates susceptibility to psychiatric disorders during development and adulthood
Nina Dedic(Chugai Pharma (United States)), Jan M. Deussing(Max Planck Institute of Psychiatry), Darina Czamara(Max Planck Institute of Psychiatry), Wolfgang Wurst(German Center for Neurodegenerative Diseases), Benedikt T. Bedenk(Max Planck Institute of Psychiatry), Max L. Pöhlmann(Harvard University), Julien Dine(Weizmann Institute of Science), Carsten T. Wotjak(Boehringer Ingelheim (Germany)), Adriana Lori(Emory University), Matthias Eder(Max Planck Institute of Psychiatry), Gerhard Rammes(Max Planck Institute of Psychiatry), Mathias V. Schmidt(Essen University Hospital), Lynn M. Almli(Centers for Disease Control and Prevention), Sven Moosmang(Boehringer Ingelheim (Germany)), Divya Mehta(Max Planck Society), Alon Chen(Max Planck Institute of Psychiatry), Kerry J. Ressler(McLean Hospital), Angela Jurik(Technical University of Munich), Julia Richter(Max Planck Institute of Psychiatry), Franz Hofmann(Pharmalog (Germany)), Meriah Metzger(Max Planck Institute of Psychiatry), Jakob Hartmann(Harvard University), Klaus V. Wagner(Max Planck Institute of Psychiatry), Elisabeth B. Binder(Max Planck Institute of Psychiatry)
Cited by 178
Related Papers
The ENIGMA Consortium: large-scale collaborative analyses of neuroimaging and genetic data
|Brain Imaging and Behavior|2014|884
Primary structure and functional expression from complementary DNA of a brain calcium channel
|Nature|1991|869
Overshadowed by the amygdala: the bed nucleus of the stria terminalis emerges as key to psychiatric disorders
|Molecular Psychiatry|2016|663