The role of PPAR alpha as a "lipostat" transcription factor.

Fatima Djouadi(Délégation Paris 5), Daniel P. Kelly(Cardiovascular Institute of the South), Carla J. Weinheimer(University of Washington)

PubMed

January 1, 1999

Cited by 12

Related Papers

Itaconate Links Inhibition of Succinate Dehydrogenase with Macrophage Metabolic Remodeling and Regulation of Inflammation

|Cell Metabolism|2016|1.5k

Peroxisome proliferator–activated receptor γ coactivator-1 promotes cardiac mitochondrial biogenesis

|Journal of Clinical Investigation|2000|1.3k

PGC-1α Deficiency Causes Multi-System Energy Metabolic Derangements: Muscle Dysfunction, Abnormal Weight Control and Hepatic Steatosis

|PLoS Biology|2005|942

A critical role for the peroxisome proliferator-activated receptor α (PPARα) in the cellular fasting response: The PPARα-null mouse as a model of fatty acid oxidation disorders

|Proceedings of the National Academy of Sciences|1999|940

The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus

|Journal of Clinical Investigation|2002|879