A critical role for the peroxisome proliferator-activated receptor α (PPARα) in the cellular fasting response: The PPARα-null mouse as a model of fatty acid oxidation disorders
Teresa C. Leone(Cardiovascular Institute of the South), Daniel P. Kelly(Washington University in St. Louis), Carla J. Weinheimer(Washington University in St. Louis)
Cited by 940
Related Papers
Peroxisome proliferator–activated receptor γ coactivator-1 promotes cardiac mitochondrial biogenesis
|Journal of Clinical Investigation|2000|1.3k
PGC-1α Deficiency Causes Multi-System Energy Metabolic Derangements: Muscle Dysfunction, Abnormal Weight Control and Hepatic Steatosis
|PLoS Biology|2005|942
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
|Journal of Clinical Investigation|2002|879
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
|Journal of Clinical Investigation|2002|765
The Failing Heart Relies on Ketone Bodies as a Fuel
|Circulation|2016|706