Profound hyperglycemia in knockout mutant mice identifies novel function for POU4F2/Brn-3b in regulating metabolic processes
Stavroula Bitsi(Imperial College London), Vishwanie Budhram‐Mahadeo(University College London)
American Journal of Physiology-Endocrinology and Metabolism
December 15, 2015
Cited by 11
Related Papers
Brn-3b enhances the pro-apoptotic effects of p53 but not its induction of cell cycle arrest by cooperating in trans-activation of bax expression
|Nucleic Acids Research|2006|38
Divergent acute versus prolonged pharmacological GLP-1R responses in adult β cell–specific β-arrestin 2 knockout mice
|Science Advances|2023|35
The POU4F2/Brn-3b transcription factor is required for the hypertrophic response to angiotensin II in the heart
|Cell Death and Disease|2019|12
Vascular dysfunction caused by loss of Brn-3b/POU4F2 transcription factor in aortic vascular smooth muscle cells is linked to deregulation of calcium signalling pathways
|Cell Death and Disease|2023|4
Divergent acute <i>versus</i> prolonged pharmacological GLP-1R responses in adult beta cell-selective β-arrestin 2 knockout mice
|bioRxiv (Cold Spring Harbor Laboratory)|2022|3