Brn-3b enhances the pro-apoptotic effects of p53 but not its induction of cell cycle arrest by cooperating in trans-activation of bax expression

Vishwanie Budhram‐Mahadeo(University College London), David S. Latchman(Laboratory of Molecular Genetics)

Nucleic Acids Research

December 1, 2006

10.1093/nar/gkl878

Cited by 38

Related Papers

Hereditary Early-Onset Parkinson's Disease Caused by Mutations in <i>PINK1</i>

|Science|2004|3.5k

The POU4F2/Brn-3b transcription factor is required for the hypertrophic response to angiotensin II in the heart

|Cell Death and Disease|2019|12

Profound hyperglycemia in knockout mutant mice identifies novel function for POU4F2/Brn-3b in regulating metabolic processes

|American Journal of Physiology-Endocrinology and Metabolism|2015|11

Vascular dysfunction caused by loss of Brn-3b/POU4F2 transcription factor in aortic vascular smooth muscle cells is linked to deregulation of calcium signalling pathways

|Cell Death and Disease|2023|4

Vascular dysfunction caused by loss of Brn-3b/POU4F2 transcription factor in aortic vascular smooth muscle cells is linked to deregulation of calcium signaling pathways.

|Research Square|2023|0