Wallerian degeneration of injured axons and synapses is delayed by a Ube4b/Nmnat chimeric gene
Till G.A. Mack(University of Cologne), Michael P. Coleman(Babraham Institute), Bogdan Beirowski(The Ohio State University Wexner Medical Center), Laura Conforti(University of Cincinnati), Monica Emanuelli(Marche Polytechnic University), Andrea Tarlton(John Radcliffe Hospital), Felipe A. Court(Pontificia Universidad Católica de Chile), V. Hugh Perry(University of Southampton), Klaus Addicks, Diana Wagner(Christian-Albrechts-Universität zu Kiel), Derek Thomson(University of Edinburgh), Richard R. Ribchester(University of Edinburgh), Febilla Fernando(Utrecht University), Christian Andressen(University of Cologne), Weiqian Mi(University of Cologne), Michael Reiner(National Association of Statutory Health Insurance Physicians), Giulio Magni(Marche Polytechnic University)
Cited by 616
Related Papers
Multidisciplinary research priorities for the COVID-19 pandemic: a call for action for mental health science
|The Lancet Psychiatry|2020|6k
The Major Risk Factors for Alzheimer’s Disease: Age, Sex, and Genes Modulate the Microglia Response to Aβ Plaques
|Cell Reports|2019|978
Wallerian degeneration: an emerging axon death pathway linking injury and disease
|Nature reviews. Neuroscience|2014|627
Regulation of Microglial Proliferation during Chronic Neurodegeneration
|Journal of Neuroscience|2013|420