Wallerian degeneration of injured axons and synapses is delayed by a Ube4b/Nmnat chimeric geneTill G.A. Mack, Michael P. Coleman, Klaus Addicks et al.|Nature Neuroscience|2001Cited by 616
A rise in NAD precursor nicotinamide mononucleotide (NMN) after injury promotes axon degenerationM. Di Stefano, Laura Conforti, Isabel Nascimento-Ferreira et al.|Cell Death and Differentiation|2014Cited by 263
WldS protein requires Nmnat activity and a short N-terminal sequence to protect axons in miceLaura Conforti, Michael P. Coleman, Anna Wilbrey et al.|The Journal of Cell Biology|2009Cited by 116
Non-Nuclear Wld <sup>S</sup> Determines Its Neuroprotective Efficacy for Axons and Synapses <i>In Vivo</i>Bogdan Beirowski, Michael P. Coleman, Elisabetta Babetto et al.|Journal of Neuroscience|2009Cited by 98
The Slow Wallerian Degeneration Protein, Wld<sup>S</sup>, Binds Directly to VCP/p97 and Partially Redistributes It within the NucleusHeike Laser, Michael P. Coleman, Laura Conforti et al.|Molecular Biology of the Cell|2005Cited by 61