Inhibition of NADPH Oxidase Prevents Advanced Glycation End Product–Mediated Damage in Diabetic Nephropathy Through a Protein Kinase C-α–Dependent Pathway
Vicki Thallas‐Bonke(Albert Einstein College of Medicine), Josephine M. Forbes(Translational Research Institute), Felicia Y. T. Yap(Baker Heart and Diabetes Institute), Melinda T. Coughlan(Baker Heart and Diabetes Institute), Mark E. Cooper(Baker Heart and Diabetes Institute), Kei Fukami(Kurume University), Leon A. Bach(Monash University), Karly C. Sourris(Baker Heart and Diabetes Institute), Sally A. Penfold(Albert Einstein College of Medicine), Suzanne R. Thorpe(University of South Carolina)
Cited by 346
Related Papers
Intensive Blood Glucose Control and Vascular Outcomes in Patients with Type 2 Diabetes
|New England Journal of Medicine|2008|7.4k
Role of oxidative stress in diabetic complications: a new perspective on an old paradigm.
|Diabetes|1999|2.4k
Diabetic kidney disease
|Nature Reviews Disease Primers|2015|1.2k
Effect of Collagen Turnover on the Accumulation of Advanced Glycation End Products
|Journal of Biological Chemistry|2000|957
The Advanced Glycation End Product, N∊-(Carboxymethyl)lysine, Is a Product of both Lipid Peroxidation and Glycoxidation Reactions
|Journal of Biological Chemistry|1996|800