GLP-1 Receptor Stimulation Reduces Amyloid-β Peptide Accumulation and Cytotoxicity in Cellular and Animal Models of Alzheimer's Disease
Yazhou Li(National Institute on Aging), Nigel H. Greig(National Institutes of Health), Kumar Sambamurti(Medical University of South Carolina), David Tweedie(National Institute on Aging), Dimitrios Kapogiannis(Office of Extramural Research), Jason A. Bailey(Indiana University School of Medicine), Mark P. Mattson(Johns Hopkins University), Debomoy K. Lahiri(Indiana University – Purdue University Indianapolis), Kara B. Duffy(University of Maryland, College Park), Balmiki Ray(Indiana University School of Medicine), TracyAnn Perry(National Institute on Aging), Harold W. Holloway(National Institute on Aging), Mary Ann Ottinger(University of Houston)
Cited by 325
Related Papers
Mitophagy inhibits amyloid-β and tau pathology and reverses cognitive deficits in models of Alzheimer’s disease
|Nature Neuroscience|2019|1.7k
Brain energy rescue: an emerging therapeutic concept for neurodegenerative disorders of ageing
|Nature Reviews Drug Discovery|2020|982
Pivotal role for neuronal Toll-like receptors in ischemic brain injury and functional deficits
|Proceedings of the National Academy of Sciences|2007|784
Calcium and neurodegeneration
|Aging Cell|2007|751
Adverse Outcome Pathway (AOP) Development I: Strategies and Principles
|Toxicological Sciences|2014|725