Polyglutamine expansion in huntingtin increases its insertion into lipid bilayers
Kimberly B. Kegel(Harvard University), Wolfgang H. Goldmann(Friedrich-Alexander-Universität Erlangen-Nürnberg), Erich E. Wanker(Max Delbrück Center), Vitali Schewkunow(Friedrich-Alexander-Universität Erlangen-Nürnberg), Marian DiFiglia(Massachusetts General Hospital), Nicholas Masso(Massachusetts General Hospital), Ellen Sapp(Massachusetts General Hospital)
Cited by 39
Related Papers
Huntingtin Expression Stimulates Endosomal–Lysosomal Activity, Endosome Tubulation, and Autophagy
|Journal of Neuroscience|2000|543
Early and Progressive Accumulation of Reactive Microglia in the Huntington Disease Brain
|Journal of Neuropathology & Experimental Neurology|2001|538
Small-molecule conversion of toxic oligomers to nontoxic β-sheet–rich amyloid fibrils
|Nature Chemical Biology|2011|442
Vinculin Promotes Cell Spreading by Mechanically Coupling Integrins to the Cytoskeleton
|Experimental Cell Research|1997|276
Feeling the forces: atomic force microscopy in cell biology
|Life Sciences|2003|218