Huntingtin Expression Stimulates Endosomal–Lysosomal Activity, Endosome Tubulation, and Autophagy
Kimberly B. Kegel(Harvard University), Marian DiFiglia(Massachusetts General Hospital)
Cited by 543
Related Papers
Early and Progressive Accumulation of Reactive Microglia in the Huntington Disease Brain
|Journal of Neuropathology & Experimental Neurology|2001|538
Huntingtin Bodies Sequester Vesicle-Associated Proteins by a Polyproline-Dependent Interaction
|Journal of Neuroscience|2004|184
Assessing average somatic CAG repeat instability at the protein level
|Scientific Reports|2019|24