PPARα suppresses insulin secretion and induces UCP2 in insulinoma cells
Karen Tordjman(Cardiovascular Research Center), Clay F. Semenkovich(Washington University in St. Louis), Daniel P. Kelly(Cardiovascular Institute of the South), Teresa C. Leone(Cardiovascular Institute of the South), Trey Coleman(Cardiovascular Research Center), Kara N. Standley(Washington University in St. Louis), Carlos Bernal‐Mizrachi(Washington University in St. Louis)
Cited by 82
Related Papers
Peroxisome proliferator–activated receptor γ coactivator-1 promotes cardiac mitochondrial biogenesis
|Journal of Clinical Investigation|2000|1.3k
PGC-1α Deficiency Causes Multi-System Energy Metabolic Derangements: Muscle Dysfunction, Abnormal Weight Control and Hepatic Steatosis
|PLoS Biology|2005|942
A critical role for the peroxisome proliferator-activated receptor α (PPARα) in the cellular fasting response: The PPARα-null mouse as a model of fatty acid oxidation disorders
|Proceedings of the National Academy of Sciences|1999|940
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
|Journal of Clinical Investigation|2002|879
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
|Journal of Clinical Investigation|2002|765