PPARα regulates cardiac energy metabolism by controlling malonyl COA concentrations in the heart
Fiona Campbell(University of Liverpool), Gary D. Lopaschuk(University of Alberta), Darrell D. Belke, Judith Y. Altarelos, Carla J. Weinheimer(University of Washington), Jason R.B. Dyck(University of Alberta), Daniel P. Kelly, Michael Courtois(Washington University in St. Louis), Alese Wagner, David L. Severson, Ray Kozak(University of Alberta)
Cited by 0
Related Papers
Itaconate Links Inhibition of Succinate Dehydrogenase with Macrophage Metabolic Remodeling and Regulation of Inflammation
|Cell Metabolism|2016|1.5k
PGC-1α Deficiency Causes Multi-System Energy Metabolic Derangements: Muscle Dysfunction, Abnormal Weight Control and Hepatic Steatosis
|PLoS Biology|2005|942
A critical role for the peroxisome proliferator-activated receptor α (PPARα) in the cellular fasting response: The PPARα-null mouse as a model of fatty acid oxidation disorders
|Proceedings of the National Academy of Sciences|1999|940
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
|Journal of Clinical Investigation|2002|879
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
|Journal of Clinical Investigation|2002|765