CD19, a Response Regulator of B Lymphocytes, Regulates Wound Healing through Hyaluronan-Induced TLR4 Signaling
Yohei Iwata(Nagasaki University), Shinichi Sato(The University of Tokyo)
Cited by 104
Related Papers
Simultaneous downregulation of KLF5 and Fli1 is a key feature underlying systemic sclerosis
|Nature Communications|2014|143
Interleukin-31 promotes fibrosis and T helper 2 polarization in systemic sclerosis
|Nature Communications|2021|79
Elevated serum interleukin-27 levels in patients with systemic sclerosis: association with T cell, B cell and fibroblast activation
|Annals of the Rheumatic Diseases|2010|60
Fli1-haploinsufficient dermal fibroblasts promote skin-localized transdifferentiation of Th2-like regulatory T cells
|Arthritis Research & Therapy|2018|40
CD147-Cyclophilin a Interactions Promote Proliferation and Survival of Cutaneous T-Cell Lymphoma
|International Journal of Molecular Sciences|2021|21