Impairment of insulin-stimulated glucose transport and ERK activation by adipocyte-specific knockout of PKC-λ produces a phenotype characterized by diminished adiposity and enhanced insulin suppression of hepatic gluconeogenesis
Mini P. Sajan(United States Department of Veterans Affairs), Robert V. Farese(University of South Florida), Gerald I. Shulman(Howard Hughes Medical Institute), Varman T. Samuels(Howard Hughes Medical Institute), Michael J Jurzak(Howard Hughes Medical Institute), Michael Leitges(Memorial University of Newfoundland), Ursula Braun(Medizinische Hochschule Hannover)
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