Muscle-specific knockout of PKC-λ impairs glucose transport and induces metabolic and diabetic syndromesRobert V. Farese, Michael Leitges, Mini P. Sajan et al.|Journal of Clinical Investigation|2007Cited by 162
AICAR and metformin, but not exercise, increase muscle glucose transport through AMPK-, ERK-, and PDK1-dependent activation of atypical PKCMini P. Sajan, Robert V. Farese, Isabelle Hainault et al.|American Journal of Physiology-Endocrinology and Metabolism|2009Cited by 96
Brain Insulin Signaling Is Increased in Insulin-Resistant States and Decreases in FOXOs and PGC-1α and Increases in Aβ1–40/42 and Phospho-Tau May Abet Alzheimer DevelopmentMini P. Sajan, Robert V. Farese, Barbara C. Hansen et al.|Diabetes|2016Cited by 87
The critical role of atypical protein kinase C in activating hepatic SREBP-1c and NFκB in obesityMini P. Sajan, Robert V. Farese, Mary L. Standaert et al.|Journal of Lipid Research|2009Cited by 55
Correction of metabolic abnormalities in a rodent model of obesity, metabolic syndrome, and type 2 diabetes mellitus by inhibitors of hepatic protein kinase C–ιMini P. Sajan, Robert V. Farese, Sonali Nimal et al.|Metabolism|2012Cited by 34