Mice deficient in BACE1, the Alzheimer's β-secretase, have normal phenotype and abolished β-amyloid generation
Yi Luo, Brad Bolon(Amgen (United States)), Steve Kahn(Amgen (United States)), Brian J. Bennett(Amgen (United States)), Safura Babu‐Khan(Amgen (United States)), Paul Denis(Amgen (United States)), Wei Fan(Amgen (United States)), Hue T. Kha(Amgen (United States)), Jianhua Zhang(Amgen (United States)), Yunhua Gong(Amgen (United States)), Laura E. Martin(Amgen (United States)), Jean-Claude Louis(Amgen (United States)), Yan Qiao(Amgen (United States)), William G. Richards(Amgen (United States)), Martin Citron(Amgen (United States)), Robert Vassar(Northwestern University)
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