Morris Schaeffer

Of the existing textbooks, none has the unique characteristics of this manual. It has been carefully planned to have breadth, yet brevity, in scope and treatment. Despite the large number of contributors, most of whom are leaders in the field, the format of each chapter is fairly uniform, albeit that some variation was obviously necessary for certain material.The introductory section gets right down to the business of describing the ways and means of identifying disease producing organisms. This is followed by sections on the identification of bacteria, fungi, parasites, viruses and rickettsia. There are also concise but ample chapters on antimicrobial agents, serodiagnosis, miscellaneous procedures and finally a section on media, reagents, and stains. Throughout, salient facts are well interspersed with informative tables and illustrations. All of these features make this book a useful and vital source of ready and authorative information.

SummaryAn epidemic of jungle fever which involved almost one-half of 400 Okinawan pioneers who were attempting to settle in the jungle rain-forest of the Bolivian lowlands adjacent to the Brazilian Matto Grosso is described. Fifteen of the cases ended fatally.A new virus, one of the Group A arthropod-borne viruses, serologically related to Semliki Forest virus, has been isolated from the blood of two of the patients. This, the Uruma virus, was responsible for 10 to 15% of the cases of jungle fever; the etiology of the remaining cases remains undetermined. Serological survey of indigenous communities reveals that this virus or a serologically related agent is endemic to the region. The Semliki Forest-Uruma-Mayaro-Chikungunja serological group of agents appears to be a world-circling group of tropical Group A arthropod-borne viruses pathogenic for man.The problem of jungle fever, or pyrexias of unknown origin, in similar attempts of nonindigenous populaces to clear and settle in tropical jungle, and the problem of multiple etiology of any epidemic that develops among them, are discussed.

Journal Article STANDARDIZATION AND CERTIFICATION OF REFERENCE ANTIGENS AND ANTISERA FOR 30 HUMAN ADENOVIRUS SEROTYPES Get access DAVID A. STEVENS, DAVID A. STEVENS Search for other works by this author on: Oxford Academic PubMed Google Scholar MORRIS SCHAEFFER, MORRIS SCHAEFFER Search for other works by this author on: Oxford Academic PubMed Google Scholar JOHN P. FOX, JOHN P. FOX Search for other works by this author on: Oxford Academic PubMed Google Scholar CARL D. BRANDT, CARL D. BRANDT Search for other works by this author on: Oxford Academic PubMed Google Scholar MARIE ROMANO MARIE ROMANO 6Microbiological Associates, Inc. of BethesdaMaryland Search for other works by this author on: Oxford Academic PubMed Google Scholar American Journal of Epidemiology, Volume 86, Issue 3, November 1967, Pages 617–633, https://doi.org/10.1093/oxfordjournals.aje.a120771 Published: 01 November 1967 Article history Received: 21 April 1967 Published: 01 November 1967

1. The procedures employed in the successful adaption of Type I (Mahoney) polimyelitis virus to Swiss mice are described. This vgirus could also infect hamsters and C3H mice but not cotton rats, while its original virulenece for monkeys was lost. 2. The use of the intraspinal route or inoculation and the selection of a mutant or variant of the virus is believed to be responsible for this adaptation. Other possible explanations are discussed. 3. With this attainment, all 3 poliomyelitis virus types have now been adapted to mice. This will permit more rapid progress in field and laboratory studies of poliomyelitis.