Smad2 and Smad3 Are Redundantly Essential for the TGF-β–Mediated Regulation of Regulatory T Plasticity and Th1 DevelopmentTomohito Takimoto, Akihiko Yoshimura, Go Muto et al.|The Journal of Immunology|2010Cited by 325
Loss of Suppressor of Cytokine Signaling 1 in Helper T Cells Leads to Defective Th17 Differentiation by Enhancing Antagonistic Effects of IFN-γ on STAT3 and SmadsKentaro Tanaka, Akihiko Yoshimura, Kenji Ichiyama et al.|The Journal of Immunology|2008Cited by 183
Selective Expansion of Foxp3-Positive Regulatory T Cells and Immunosuppression by Suppressors of Cytokine Signaling 3-Deficient Dendritic CellsYumiko Matsumura, Akihiko Yoshimura, Takashi Kobayashi et al.|The Journal of Immunology|2007Cited by 105
Correction: Smad2 and Smad3 Are Redundantly Essential for the TGF-β–Mediated Regulation of Regulatory T Plasticity and Th1 DevelopmentTomohito Takimoto, Akihiko Yoshimura, Yu Wakabayashi et al.|The Journal of Immunology|2010Cited by 53