Muscle-specific knockout of PKC-λ impairs glucose transport and induces metabolic and diabetic syndromesRobert V. Farese, Michael Leitges, Mini P. Sajan et al.|Journal of Clinical Investigation|2007Cited by 162
AICAR and metformin, but not exercise, increase muscle glucose transport through AMPK-, ERK-, and PDK1-dependent activation of atypical PKCMini P. Sajan, Robert V. Farese, Gautam Bandyopadhyay et al.|American Journal of Physiology-Endocrinology and Metabolism|2009Cited by 96
The critical role of atypical protein kinase C in activating hepatic SREBP-1c and NFκB in obesityMini P. Sajan, Robert V. Farese, Mary L. Standaert et al.|Journal of Lipid Research|2009Cited by 55
Correction of metabolic abnormalities in a rodent model of obesity, metabolic syndrome, and type 2 diabetes mellitus by inhibitors of hepatic protein kinase C–ιMini P. Sajan, Robert V. Farese, Sonali Nimal et al.|Metabolism|2012Cited by 34
Combined thiazolidinedione–metformin treatment synergistically improves insulin signalling to insulin receptor substrate-1-dependent phosphatidylinositol 3-kinase, atypical protein kinase C and protein kinase B/Akt in human diabetic muscleN. Temofonte, Robert V. Farese, Mini P. Sajan et al.|Diabetologia|2008Cited by 15