Interaction of ARC and Daxx: A Novel Endogenous Target to Preserve Motor Function and Cell Loss after Focal Brain Ischemia in MiceStefan Donath, Christoph Harms, Matthias Endres et al.|Journal of Neuroscience|2016Cited by 34
Delayed administration of TAT.ARC protein attenuates focal ischemic brain injury and fosters recovery in the long-term: behavioral data of neuroscores and rota rod tests, daily temperature and body weight measuresStefan Donath, Christoph Harms, Junfeng An et al.|Figshare|2016Cited by 1
Dose response curve shows better survival of TAT.ARC protein transduced primary cortical neurons in comparison to TAT beta galactosidase transduced neurons after oxygen glucose deprivationStefan Donath, Christoph Harms, Junfeng An et al.|Figshare|2016Cited by 1
TAT.ARC protein administration in the contralateral ventricle decreased lesion volume in subregions of the ischemic territory after 24 h and 72 h after 60 min middle cerebral artery occlusionStefan Donath, Christoph Harms, Junfeng An et al.|Figshare|2016Cited by 1
Striatal volume and ventricle area was assessed in NeuN DAB sections at 28 days after 30 min MCAo in sham and MCAo mice treated with TAT proteins 3 hours after the onset of ischemia.Stefan Donath, Christoph Harms, Junfeng An et al.|Figshare|2016Cited by 1