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Cardiac Fibroblasts Contribute to Myocardial Dysfunction in Mice with Sepsis: The Role of NLRP3 Inflammasome ActivationWenbo Zhang, Tao Rui, Xuemei Xu et al.|PLoS ONE|2014Cited by 105
Carbon monoxide releasing molecule-3 improves myocardial function in mice with sepsis by inhibiting NLRP3 inflammasome activation in cardiac fibroblastsWenbo Zhang, Tao Rui, Aibin Tao et al.|Basic Research in Cardiology|2017Cited by 101
The Alarmin Cytokine, High Mobility Group Box 1, Is Produced by Viable Cardiomyocytes and Mediates the Lipopolysaccharide-Induced Myocardial Dysfunction via a TLR4/Phosphatidylinositol 3-Kinase γ PathwayHu Xu, Tao Rui, Zhaoliang Su et al.|The Journal of Immunology|2009Cited by 96