Pregnancy-Enhanced Endothelial Nitric Oxide Synthase (eNOS) Activation in Uterine Artery Endothelial Cells Shows Altered Sensitivity to Ca<sup>2+</sup>, U0126, and Wortmannin But Not LY294002—Evidence that Pregnancy Adaptation of eNOS Activation Occurs at Multiple Levels of Cell SignalingJeremy A. Sullivan, Ian M. Bird|Endocrinology|2006Cited by 30
Inhibition of MEK/ERK1/2 signalling alters endothelial nitric oxide synthase activity in an agonist-dependent mannerJacqueline M. Cale, Ian M. Bird|Biochemical Journal|2006Cited by 28
Pregnancy-enhanced store-operated Ca2+ channel function in uterine artery endothelial cells is associated with enhanced agonist-specific transient receptor potential channel 3-inositol 1,4,5-trisphosphate receptor 2 interactionShannon M. Gifford, Ian M. Bird|Journal of Endocrinology|2006Cited by 28