Neurotoxicity and Memory Deficits Induced by Soluble Low-Molecular-Weight Amyloid- 1-42 Oligomers Are Revealed In Vivo by Using a Novel Animal ModelJonathan Brouillette, Luc Buée|Journal of Neuroscience|2012Cited by 171
Neuroligin-1 is altered in the hippocampus of Alzheimer’s disease patients and mouse models, and modulates the toxicity of amyloid-beta oligomersJulien Dufort‐Gervais, Jonathan Brouillette|Scientific Reports|2020Cited by 50
Hippocampal injections of soluble amyloid-beta oligomers alter electroencephalographic activity during wake and slow-wave sleep in ratsAudrey Hector, Jonathan Brouillette|Alzheimer s Research & Therapy|2023Cited by 13
Evidence for a GABA<sub>B</sub> receptor component in the spinal action of Substance P (SP) on arterial blood pressure in the awake ratJonathan Brouillette, Réjean Couture|British Journal of Pharmacology|2002Cited by 10
Upregulation of tachykinin NK‐1 and NK‐3 receptor binding sites in the spinal cord of spontaneously hypertensive rat: impact on the autonomic control of blood pressureFrank Cloutier, Réjean Couture, Kathleen Deschamps et al.|British Journal of Pharmacology|2006Cited by 9