Lack of NLRP3-inflammasome leads to gut-liver axis derangement, gut dysbiosis and a worsened phenotype in a mouse model of NAFLDI. Pierantonelli, Gianluca Svegliati‐Baroni, Claudio Pinto et al.|Scientific Reports|2017Cited by 83
Author Correction: Lack of NLRP3-inflammasome leads to gut-liver axis derangement, gut dysbiosis and a worsened phenotype in a mouse model of NAFLDI. Pierantonelli, Gianluca Svegliati‐Baroni, C. Rychlicki et al.|Scientific Reports|2017Cited by 8
Lack of NLRP3-Inflammasome Leads to Gut-Liver AXIS Derangement and Increases Hepatic Injury in a Mouse Model of Non-Alcoholic Fatty Liver DiseaseI. Pierantonelli, Gianluca Svegliati‐Baroni, L. Agostinelli et al.|Journal of Hepatology|2016Cited by 1
Gut–liver axis derangement due to lack of inflammasome activity leads to visceral obesity and NASH developmentL. Agostinelli, Gianluca Svegliati‐Baroni, C. Rychlicki et al.|Digestive and Liver Disease|2015Cited by 0
Lack of NLRP3-inflammasome leads to gut-liver axis derangement and increases hepatic injury in a mouse model of NAFLDI. Pierantonelli, Gianluca Svegliati‐Baroni, L. Agostinelli et al.|Digestive and Liver Disease|2016Cited by 0