Pancreatic cell plasticity and cancer initiation induced by oncogenic Kras is completely dependent on wild-type PI 3-kinase p110αRomain Baer, Julie Guillermet‐Guibert, Barbara Garmy‐Susini et al.|Genes & Development|2014Cited by 127
Loss of Somatostatin Receptor Subtype 2 Promotes Growth of KRAS-Induced Pancreatic Tumors in Mice by Activating PI3K Signaling and Overexpression of CXCL16Mounira Chalabi‐Dchar, Corinne Bousquet, Stéphanie Cassant‐Sourdy et al.|Gastroenterology|2015Cited by 46
Essential Interaction of Egr-1 at an Islet-specific Response Element for Basal and Gastrin-dependent Glucagon Gene Transactivation in Pancreatic α-CellsStéphane Leung–Theung–Long, Marlène Dufresne|Journal of Biological Chemistry|2004Cited by 27
311 Somatostatin Receptor SST2 Haploinsufficiency Accelerates KRASG12d-Initiated Pancreatic Carcinogenesis by Promoting InflammationMounira Chalabi, Corinne Bousquet, Stéphanie Cassant‐Sourdy et al.|Gastroenterology|2013Cited by 0
The non-mutated PI3Kalpha is a master signaling enzyme for pancreatic cancerogenesisRomain Baer, Julie Guillermet‐Guibert, Marlène Dufresne et al.|Pancreatology|2014Cited by 0