Murine Sialidase Neu3 facilitates GM2 degradation and bypass in mouse model of Tay-Sachs diseaseVolkan Seyrantepe, Taeko Miyagi, Emin Öztaş et al.|Experimental Neurology|2017Cited by 82
GM2 ganglioside accumulation causes neuroinflammation and behavioral alterations in a mouse model of early onset Tay-Sachs diseaseSeçil Akyıldız Demir, Volkan Seyrantepe|Journal of Neuroinflammation|2020Cited by 65
Sialidase neu4 deficiency is associated with neuroinflammation in miceZehra Kevser Timur, Volkan Seyrantepe, Orhan Kerim İnci et al.|Glycoconjugate Journal|2021Cited by 8
Silencing of B4Galnt1 Gene Prevents GM2 Accumulation in Tay-Sachs CellsNurselin Ateş, Volkan Seyrantepe, Seçil Akyıldız Demir et al.|Advances in Cell and Gene Therapy|2024Cited by 1