PKA Phosphorylation of NCLX Reverses Mitochondrial Calcium Overload and Depolarization, Promoting Survival of PINK1-Deficient Dopaminergic NeuronsMarko Kostić, Israel Sekler, Marthe H. R. Ludtmann et al.|Cell Reports|2015Cited by 173
Allosteric Regulation of NCLX by Mitochondrial Membrane Potential Links the Metabolic State and Ca2+ Signaling in MitochondriaMarko Kostić, Israel Sekler, Tomer Katoshevski|Cell Reports|2018Cited by 86
LRRK2 deficiency induced mitochondrial Ca2+ efflux inhibition can be rescued by Na+/Ca2+/Li+ exchanger upregulationMarthe H. R. Ludtmann, Andrey Y. Abramov, Marko Kostić et al.|Cell Death and Disease|2019Cited by 83
Aberrant activity of mitochondrial NCLX is linked to impaired synaptic transmission and is associated with mental retardationAlexandra Stavsky, Israel Sekler, Tomer Katoshevsky et al.|Communications Biology|2021Cited by 55
Functional properties and mode of regulation of the mitochondrial Na+/Ca2+ exchanger, NCLXMarko Kostić, Israel Sekler|Seminars in Cell and Developmental Biology|2019Cited by 54