Dietary cholesterol, rather than liver steatosis, leads to hepatic inflammation in hyperlipidemic mouse models of nonalcoholic steatohepatitis†Kristiaan Wouters, Marten H. Hofker, Patrick J. van Gorp et al.|Hepatology|2008Cited by 467
Early diet-induced non-alcoholic steatohepatitis in APOE2 knock-in mice and its prevention by fibratesRonit Shiri‐Sverdlov, Marten H. Hofker, Kristiaan Wouters et al.|Journal of Hepatology|2005Cited by 241
LDL Receptor Knock-Out Mice Are a Physiological Model Particularly Vulnerable to Study the Onset of Inflammation in Non-Alcoholic Fatty Liver DiseaseVeerle Bieghs, Ronit Shiri‐Sverdlov, Tim Hendrikx et al.|PLoS ONE|2012Cited by 174
Understanding hyperlipidemia and atherosclerosis: lessons from genetically modified apoe and ldlr miceKristiaan Wouters, Marten H. Hofker, Ronit Shiri‐Sverdlov et al.|Clinical Chemistry and Laboratory Medicine (CCLM)|2005Cited by 166
Role of Scavenger Receptor A and CD36 in Diet-Induced Nonalcoholic Steatohepatitis in Hyperlipidemic MiceVeerle Bieghs, Ronit Shiri‐Sverdlov, Kristiaan Wouters et al.|Gastroenterology|2010Cited by 160