A novel P53/<scp>POMC</scp>/Gαs/<scp>SASH</scp>1 autoregulatory feedback loop activates mutated <scp>SASH</scp>1 to cause pathologic hyperpigmentationDing’an Zhou, Qinghe Xing, Zhiyun Wei et al.|Journal of Cellular and Molecular Medicine|2016Cited by 26
Mutated SASH1 promotes Mitf expression in a heterozygous mutated SASH1 knock‑in mouse modelZexi Xu, Ding’an Zhou|International Journal of Molecular Medicine|2020Cited by 13
The circadian clock gene, BMAL1, promotes radiosensitization in nasopharyngeal carcinoma by inhibiting the epithelial-to-mesenchymal transition via the TGF-β1/Smads/Snail1 axisYuxin Li, Feng Jin, Kai Shang et al.|Oral Oncology|2024Cited by 10
Blockade of a novel MAP4K4-LATS2-SASH1-YAP1 cascade inhibits tumorigenesis and metastasis in luminal breast cancerPingping Yang, Ding’an Zhou|Journal of Biological Chemistry|2024Cited by 7
The PER3 rs772027021 SNP induces pigmentation phenotypes of dyschromatosis universalis hereditariaHongyu Chen, Ding’an Zhou|Research Square|2022Cited by 2