G protein–coupled receptors activate p38 MAPK via a non-canonical TAB1–TAB2– and TAB1–TAB3–dependent pathway in endothelial cellsNeil Grimsey, JoAnn Trejo|Journal of Biological Chemistry|2019Cited by 47
Recycling and Endosomal Sorting of Protease-activated Receptor-1 Is Distinctly Regulated by Rab11A and Rab11B ProteinsNeil Grimsey, JoAnn Trejo|Journal of Biological Chemistry|2015Cited by 41
Characterization of Thrombin-Bound Dabigatran Effects on Protease-Activated Receptor-1 Expression and Signaling In VitroBuxin Chen, JoAnn Trejo|Molecular Pharmacology|2015Cited by 31
α-Arrestin ARRDC3 tumor suppressor function is linked to GPCR-induced TAZ activation and breast cancer metastasisAleena Arakaki, JoAnn Trejo|Journal of Cell Science|2021Cited by 30
An siRNA library screen identifies CYLD and USP34 as deubiquitinases that regulate GPCR-p38 MAPK signaling and distinct inflammatory responsesNorton Cheng, JoAnn Trejo|Journal of Biological Chemistry|2023Cited by 8