Occurrence and co-localization of amyloid β-protein and apolipoprotein E in perivascular drainage channels of wild-type and APP-transgenic miceDietmar Rudolf Thal, Estibaliz Capetillo‐Zarate, Sergey Larionov et al.|Neurobiology of Aging|2006Cited by 57
β-Site Amyloid Precursor Protein Cleaving Enzyme 1 Increases Amyloid Deposition in Brain Parenchyma but Reduces Cerebrovascular Amyloid Angiopathy in Aging BACE × APP[V717I] Double-Transgenic MiceMichael Willem, Fred Van Leuven, Peter Borghgraef et al.|American Journal Of Pathology|2004Cited by 55
Neuronal or Glial Expression of Human Apolipoprotein E4 Affects Parenchymal and Vascular Amyloid Pathology Differentially in Different Brain Regions of Double- and Triple-Transgenic MiceTom Van Dooren, Fred Van Leuven, David Muyllaert et al.|American Journal Of Pathology|2006Cited by 34
Pathological Hallmarks, Clinical Parallels, and Value for Drug Testing in Alzheimer's Disease of the APP[V717I] London Transgenic Mouse ModelAn Tanghe, Tom Van Dooren, Annelies Termont et al.|International Journal of Alzheimer s Disease|2010Cited by 30
O2‐03‐03: Phenotypic and genetic subclassification of Alzheimer's diseaseDietmar Rudolf Thal, Estifanos Ghebremedhin, Heike Koelsch et al.|Alzheimer s & Dementia|2009Cited by 20