NO-induced regulation of human trabecular meshwork cell volume and aqueous humor outflow facility involve the BK<sub>Ca</sub> ion channelW. Michael Dismuke, Dorette Z. Ellis, C. Mbadugha|American Journal of Physiology-Cell Physiology|2008Cited by 114
Endogenous Regulation of Human Schlemm's Canal Cell Volume by Nitric Oxide SignalingDorette Z. Ellis, W. Michael Dismuke, Najam A. Sharif|Investigative Ophthalmology & Visual Science|2010Cited by 80
Characterization of Soluble Guanylate Cyclase in NO-Induced Increases in Aqueous Humor Outflow Facility and in the Trabecular MeshworkDorette Z. Ellis, Binna Chokshi, W. Michael Dismuke|Investigative Ophthalmology & Visual Science|2009Cited by 68
Human Trabecular Meshwork Cell Volume Decrease by NO-Independent Soluble Guanylate Cyclase Activators YC-1 and BAY-58-2667 Involves the BK<sub>Ca</sub>Ion ChannelW. Michael Dismuke, Dorette Z. Ellis, Najam A. Sharif|Investigative Ophthalmology & Visual Science|2009Cited by 33
Activation of the BK <sub>Ca</sub> Channel Increases Outflow Facility and Decreases Trabecular Meshwork Cell VolumeW. Michael Dismuke, Dorette Z. Ellis|Journal of Ocular Pharmacology and Therapeutics|2009Cited by 32