Loss of filamentous actin, tight junction protein expression, and paracellular barrier integrity in frataxin-deficient human brain microvascular endothelial cells—implications for blood-brain barrier physiology in Friedreich’s ataxiaFrances M. Smith, Daniel J. Kosman|Frontiers in Molecular Biosciences|2024Cited by 3
Frataxin-deficient human brain microvascular endothelial cells lose polymerized actin and are paracellularly permeable –implications for blood-brain barrier integrity in Friedreich’s AtaxiaFrances M. Smith, Daniel J. Kosman|bioRxiv (Cold Spring Harbor Laboratory)|2023Cited by 2
Brain microvascular endothelial cells differentiated from a Friedreich’s Ataxia patient iPSC are deficient in tight junction protein expression and paracellularly permeableFrances M. Smith, Daniel J. Kosman|Frontiers in Molecular Neuroscience|2025Cited by 1
Decreased filamentous actin and tight junction protein expression, and paracellular permeability in Frataxin-deficient human brain microvascular endothelial cells – implications for blood-brain barrier integrity in Friedreich's AtaxiaFrances M. Smith, Daniel J. Kosman|Research Square|2023Cited by 0