Postconditioning via stuttering reperfusion limits myocardial infarct size in rabbit hearts: role of ERK1/2Chad E. Darling, Karin Przyklenk, Michelle Maynard et al.|American Journal of Physiology-Heart and Circulatory Physiology|2005Cited by 199
Aging Mouse Hearts Are Refractory to Infarct Size Reduction With Post-ConditioningKarin Przyklenk, Peter Whittaker, Michelle Maynard et al.|Journal of the American College of Cardiology|2008Cited by 126
Cardioprotection with Postconditioning: Loss of Efficacy in Murine Models of Type-2 and Type-1 DiabetesKarin Przyklenk, Peter Whittaker, Michelle Maynard et al.|Antioxidants and Redox Signaling|2010Cited by 123
Pretreatment with d-myo-Inositol Trisphosphate Reduces Infarct Size in Rabbit Hearts: Role of Inositol Trisphosphate Receptors and Gap Junctions in Triggering ProtectionKarin Przyklenk, Peter Whittaker, Chad E. Darling et al.|Journal of Pharmacology and Experimental Therapeutics|2005Cited by 23
First molecular evidence that inositol trisphosphate signaling contributes to infarct size reduction with preconditioningKarin Przyklenk, Peter Whittaker, Michelle Maynard|American Journal of Physiology-Heart and Circulatory Physiology|2006Cited by 10