A mechanism of resistance to gefitinib mediated by cellular reprogramming and the acquisition of an FGF2-FGFR1 autocrine growth loopK E Ware, Lynn E. Heasley|Oncogenesis|2013Cited by 256
Rapidly Acquired Resistance to EGFR Tyrosine Kinase Inhibitors in NSCLC Cell Lines through De-Repression of FGFR2 and FGFR3 ExpressionKathryn E. Ware, Lynn E. Heasley|PLoS ONE|2010Cited by 145
Differential Gene Regulation by Specific Gain-of-function JNK1 Proteins Expressed in Swiss 3T3 FibroblastsSun‐Young Han, Lynn E. Heasley|Journal of Biological Chemistry|2002Cited by 48
A Receptor Tyrosine Kinase Network Composed of Fibroblast Growth Factor Receptors, Epidermal Growth Factor Receptor, v-erb-b2 Erythroblastic Leukemia Viral Oncogene Homolog 2, and Hepatocyte Growth Factor Receptor Drives Growth and Survival of Head and Neck Squamous Carcinoma Cell LinesKatherine R. Singleton, Lynn E. Heasley|Molecular Pharmacology|2013Cited by 43
A tyrosine kinase inhibitor-induced interferon response positively associates with clinical response in EGFR-mutant lung cancerNatalia J. Gurule, Lynn E. Heasley|npj Precision Oncology|2021Cited by 40