Immunoglobulin Activation of T Cell Chemoattractant Expression in Fibroblasts from Patients with Graves’ Disease Is Mediated Through the Insulin-Like Growth Factor I Receptor PathwayJane Pritchard, Terry J. Smith, William W. Cruikshank et al.|The Journal of Immunology|2003Cited by 294
Igs from Patients with Graves’ Disease Induce the Expression of T Cell Chemoattractants in Their FibroblastsJane Pritchard, Terry J. Smith, Noah Horst et al.|The Journal of Immunology|2002Cited by 171
Cutting Edge: IL-16/CD4 Preferentially Induces Th1 Cell Migration: Requirement of CCR5Elizabeth Lynch, William W. Cruikshank, Claudia A. W. Heijens et al.|The Journal of Immunology|2003Cited by 123
Synovial Fibroblasts from Patients with Rheumatoid Arthritis, Like Fibroblasts from Graves’ Disease, Express High Levels of IL-16 When Treated with Igs against Insulin-Like Growth Factor-1 ReceptorJane Pritchard, Terry J. Smith, Shanli Tsui et al.|The Journal of Immunology|2004Cited by 73
Neutralization of Interleukin-16 Protects Nonobese Diabetic Mice From Autoimmune Type 1 Diabetes by a CCL4-Dependent MechanismCraig Meagher, Terry L. Delovitch, Josh Beilke et al.|Diabetes|2010Cited by 44