The Amyloid-β Oligomer Hypothesis: Beginning of the Third DecadeErika N. Cline, William L. Klein|Journal of Alzheimer s Disease|2018Cited by 854
<i>N</i>‐Methyl‐<scp>d</scp>‐aspartate receptors are required for synaptic targeting of Alzheimer’s toxic amyloid‐β peptide oligomersHelena Decker, Sérgio T. Ferreira, Sofia Jürgensen et al.|Journal of Neurochemistry|2010Cited by 162
Salvianolic acid B inhibits Aβ fibril formation and disaggregates preformed fibrils and protects against Aβ-induced cytotoxictySiva Sundara Kumar Durairajan, Min Li, William L. Klein et al.|Neurochemistry International|2007Cited by 149
Altered succinylation of mitochondrial proteins, APP and tau in Alzheimer’s diseaseYun Yang, Gary E. Gibson, Elizabeth T. Anderson et al.|Nature Communications|2022Cited by 109
An Essential Role for Alzheimer’s-Linked Amyloid Beta Oligomers in Neurodevelopment: Transient Expression of Multiple Proteoforms during Retina HistogenesisSamuel C. Bartley, William L. Klein|International Journal of Molecular Sciences|2022Cited by 12