Tenascin-C promotes chronic pressure overload-induced cardiac dysfunction, hypertrophy and myocardial fibrosisBruno K. Podesser, Attila Kiss, Christoph Kaun et al.|Journal of Hypertension|2017Cited by 52
Tenascin-C Aggravates Ventricular Dilatation and Angiotensin-Converting Enzyme Activity After Myocardial Infarction in MiceDavid Santer, Bruno K. Podesser, Felix Nagel et al.|ESC Heart Failure|2020Cited by 26
The fourth report of the European Registry for Patients with Mechanical Circulatory Support (EUROMACS) of the European Association for Cardiothoracic Surgery: focus on standardized outcome ratiosKevin M. Veen, Felix Schoenrath, Christoffer Stark et al.|European Journal of Cardio-Thoracic Surgery|2025Cited by 18
The impact of age on cardiac function and extracellular matrix component expression in adverse post-infarction remodeling in miceFelix Nagel, Bruno K. Podesser, David Santer et al.|Experimental Gerontology|2019Cited by 11
P100Tenascin-C is induced by chronic hypoxia in cardiomyoblasts and exacerbates post myocardial infarction remodelingDavid Santer, Bruno K. Podesser, Inês Gonçalves et al.|Cardiovascular Research|2018Cited by 0