Gpr17 in AgRP Neurons Regulates Feeding and Sensitivity to Insulin and LeptinHongxia Ren, Domenico Accili|Diabetes|2015Cited by 51
Glut4 expression defines an insulin-sensitive hypothalamic neuronal populationHongxia Ren, Domenico Accili, Taylor Y. Lu et al.|Molecular Metabolism|2014Cited by 35
Intestinal Gpr17 deficiency improves glucose metabolism by promoting GLP-1 secretionShijun Yan, Hongxia Ren, Jason M. Conley et al.|Cell Reports|2022Cited by 26
Gpr17 deficiency in POMC neurons ameliorates the metabolic derangements caused by long-term high-fat diet feedingAustin M. Reilly, Hongxia Ren, Shudi Zhou et al.|Nutrition and Diabetes|2019Cited by 24
Human GPR17 missense variants identified in metabolic disease patients have distinct downstream signaling profilesJason M. Conley, Hongxia Ren|Journal of Biological Chemistry|2021Cited by 9