Physiological Mitochondrial Fragmentation Is a Normal Cardiac Adaptation to Increased Energy DemandMichael Coronado, Daniel Bernstein, Roberta A. Gottlieb et al.|Circulation Research|2017Cited by 143
Hypertrophic cardiomyopathy β-cardiac myosin mutation (P710R) leads to hypercontractility by disrupting super relaxed stateAlison Schroer Vander Roest, Daniel Bernstein, Chao Liu et al.|Proceedings of the National Academy of Sciences|2021Cited by 81
Time‐dependent evolution of functional <i>vs.</i> remodeling signaling in induced pluripotent stem cell‐derived cardiomyocytes and induced maturation with biomechanical stimulationGwanghyun Jung, Daniel Bernstein, Giovanni Fajardo et al.|The FASEB Journal|2015Cited by 68
miR-21 is associated with fibrosis and right ventricular failureSushma Reddy, Daniel Bernstein, Dong‐Qing Hu et al.|JCI Insight|2017Cited by 44
Hypertrophic cardiomyopathy ß-cardiac myosin mutation (P710R) leads to hypercontractility by disrupting super-relaxed stateAlison Schroer Vander Roest, Daniel Bernstein, Chao Liu et al.|bioRxiv (Cold Spring Harbor Laboratory)|2020Cited by 4