TREK-1 (K2P2.1) K+ channels are suppressed in patients with atrial fibrillation and heart failure and provide therapeutic targets for rhythm controlPatrick Lugenbiel, Dierk Thomas, Katharina Govorov et al.|Basic Research in Cardiology|2016Cited by 48
Atrial Fibrillation Complicated by Heart Failure Induces Distinct Remodeling of Calcium Cycling ProteinsPatrick Lugenbiel, Dierk Thomas, Katharina Govorov et al.|PLoS ONE|2015Cited by 42
Inhibition of Histone Deacetylases Induces K+ Channel Remodeling and Action Potential Prolongation in HL-1 Atrial CardiomyocytesPatrick Lugenbiel, Dierk Thomas, Ann‐Kathrin Rahm et al.|Cellular Physiology and Biochemistry|2018Cited by 21
Epigenetic regulation of cardiac electrophysiology in atrial fibrillation: HDAC2 determines action potential duration and suppresses NRSF in cardiomyocytesPatrick Lugenbiel, Dierk Thomas, Fadwa A. El Tahry et al.|Basic Research in Cardiology|2021Cited by 17
Atrial myofibroblast activation and connective tissue formation in a porcine model of atrial fibrillation and reduced left ventricular functionPatrick Lugenbiel, Dierk Thomas, Katharina Govorov et al.|Life Sciences|2017Cited by 16